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ILAE Announces Journal Awards

The ILAE awards several journal prizes each year. The Epilepsia Clinical Science Prize and the Epilepsia Basic Science Prize are awarded to the first authors of original research articles published in Epilepsia which have made the most significant contribution to advancing knowledge and understanding in clinical and basic science in the field of epilepsy, respectively. The Epileptic Disorders Educational Prize is awarded to the paper published in Epileptic Disorders in the previous year which has made the most significant contribution to the educational mission of the journal. See below details of the prizes and interviews with the recipients.

Saadet Mercimek-Mahmutoglu Lena Nguyen Ioana Mindruta

Epilepsia Clinical Science
Saadet Mercimek-Mahmutoglu
Canada

Epilepsia Basic Science
Lena H. Nguyen
USA

Epileptic Disorders
Educational Prize
Ioana Mindruta
Romania

News from ILAE All news

Apply now for 2017 Leadership Development Program

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The ILAE is offering 20 Leadership Development Awards to young physician leaders selected from ILAE chapters around the world. It affords a unique and exclusive opportunity to receive leadership development training and occasions to network with other rising leaders.


EpilepsyDiagnosis.org now includes structural etiologies of the epilepsies

The structural etiology section of the EpilepsyDiagnosis.org diagnostic manual has been expanded to include details of major structural brain abnormalities that cause epilepsy, including the clinical context in which they occur, seizure types seen, EEG features, imaging features, genetic contributions, and differential diagnosis.

NEW Epilepsy Classification Roadmap
Deadline to submit comments extended to 11 September!

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The Classification of the Epilepsies moves forward – a roadmap for the development of an updated Classification is featured in Epilepsia Open. We present a framework to approach classification of patients in the clinic together with new concepts and terminology. We invite your comments for further refinement and consideration as we develop a Classification that will serve in clinical practice for the foreseeable future.


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News Around the World All news

Farewell: Uwe Heinemann

Uwe Heinemann

The epilepsy community lost one of its leading researchers and mentors with the death of Uwe Heinemann on September 8, 2016. At the time of his death, Uwe was a Senior Professor at the Charité in Berlin where he had been a Professor of Neurophysiology and Neuroscience for the last 23 years. He was a leading and passionate researcher in epilepsy with an insatiable desire to understand its causes and the causes of drug resistance. He also studied the brain circuits that support seizures, and in those studies helped us to understand how the brain itself works. More …


Advance notice of Trobalt®/Potiga® discontinuation

GlaxoSmithKline has made the decision to discontinue Trobalt/Potiga (retigabine/ezogabine) from all markets in which the product is currently available. Trobalt/Potiga will remain available to existing epilepsy patients until the end of June 2017 to enable identification of an appropriate alternative where required, after which the license will be withdrawn. In the meantime HCPs are being advised to start transitioning their patients off the medicine.


Swiss Epilepsy League supports innovative research project
Epilepsie-Liga unterstützt innovatives Forschungsprojekt

Jean-Marc Fritschy and Tilo Gschwind
Jean-Marc Fritschy (left) and Tilo Gschwind

The Swiss League Against Epilepsy announces Prof. Jean-Marc Fritschy and Tilo Gschwind of the University of Zurich have been awarded this year's Research Grant of CHF 25,000. With the help of innovative optogenetics, the researchers are investigating the development of epilepsy and paving the way for new treatment methods. Applications are now open for next year's grant for start-up funding for research projects for the advancement of experimental or clinical research in the field of epileptology.

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Clinical Research News All news

Surgical treatment of pediatric focal cortical dysplasia: Clinical spectrum and surgical outcome.

Researchers analyzed the clinical presentation and outcomes of surgically treated focal cortical dysplasia (FCD) in children and concluded that FCD can cause FE and EE in pediatric age, and resective surgery should be considered as a treatment option for both types of epilepsy. Neurology vol. 87 no. 9, 945-95.


New diagnostic criteria for neurocysticercosis: Reliability and validity

The diagnosis of neurocysticercosis (NCC) remains problematic due to the heterogeneity of its clinical, immunological, and imaging characteristics. A group of Latin American NCC experts developed by consensus a new set of diagnostic criteria for NCC. A multicenter, retrospective study was then conducted to validate it. These criteria have acceptable reliability and validity and could be a new tool for clinicians and researchers. Annals of Neurology DOI: 10.1002/ana.24732

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Basic Science of Epilepsy News All news

Tight Coupling of Astrocyte pH Dynamics to Epileptiform Activity Revealed by Genetically Encoded pH Sensors

Dynamic changes in intracellular ion concentrations are central to the initiation and progression of epileptic seizures. However, it is not known how changes in intracellular H+ concentration (ie, pH) differ between different cell types during seizures. Using recently developed pH-sensitive proteins, we demonstrate that astrocytes undergo rapid alkalinization during periods of seizure-like activity, which is in stark contrast to the acidification that occurs in neighboring neurons. (doi: 10.1523/JNEUROSCI.0664-16.2016)

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Reducing premature KCC2 expression rescues seizure susceptibility and spine morphology in atypical febrile seizures

Atypical febrile seizures are considered a risk factor for epilepsy onset and cognitive impairments later in life. Patients with temporal lobe epilepsy and a history of atypical febrile seizures often carry a cortical malformation. This association has led to the hypothesis that the presence of a cortical dysplasia exacerbates febrile seizures in infancy, in turn increasing the risk for neurological sequelae. The authors' findings demonstrate that KCC2 overexpression in a compromised developing brain increases febrile seizure susceptibility and contribute to dendritic spine alterations. (Neurobiology of Disease doi:10.1016/j.nbd.2016.02.014)

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